Basal ganglia T2 hyperintensity

Last revised by Jack Garnham on 19 Apr 2024

Citation, DOI, disclosures and article data

Citation:

Gaillard F, Garnham J, Sattouf M, et al. Basal ganglia T2 hyperintensity. Reference article, Radiopaedia.org (Accessed on 20 Apr 2024) https://doi.org/10.53347/rID-969

DOI:

https://doi.org/10.53347/rID-969

Permalink:

https://radiopaedia.org/articles/969?iframe=true

rID:

969

Article created:

2 May 2008, Frank Gaillard ◉ ◈

Disclosures:

At the time the article was created Frank Gaillard had no recorded disclosures.

View Frank Gaillard's current disclosures

Last revised:

19 Apr 2024, Jack Garnham

Disclosures:

At the time the article was last revised Jack Garnham had no financial relationships to ineligible companies to disclose.

View Jack Garnham's current disclosures

Revisions:

19 times, by 10 contributors - see full revision history and disclosures

Systems:

Central Nervous System

Sections:

Gamuts

Tags:

refs

Synonyms:

Increased T2 signal in the basal ganglia
Basal ganglia T2 hypersignal

The causes of basal ganglia T2 hyperintensity can be remembered using the mnemonic LINT:

lymphoma
ischemia
- hypoxic-ischemic encephalopathy
- venous infarction (internal cerebral vein thrombosis)
neurodegenerative
- autoimmune encephalitis (e.g. anti-D2 dopamine receptor / anti-CV2 encephalitis) ²
- Creutzfeldt-Jakob disease
metabolic
- extrapontine myelinolysis
- Huntington disease: especially caudate heads
- methylmalonic acidemia
- mitochondrial diseases (e.g. Leigh disease)
- Wilson disease
- hepatic encephalopathy
- uremic encephalopathy
- hypoglycemic encephalopathy
toxins
infections
- flavivirus encephalitis
  - Japanese encephalitis
- rabies encephalitis
others
- focal areas of signal intensity (fasi) in neurofibromatosis type 1

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