Complications following gastric banding
There are many complications that can occur following gastric banding. It is helpful to divide these into early and late post-surgical complications.
Although the exact mode of presentation can vary depending on the underlying complication common modes of presentation that lead to imaging evaluation include abdominal pain, retching and vomiting.
- chronic gastric band erosion
- gastric band slippage / eccentric pouch dilatation
- pouch dilatation / gastric stomal stenosis
port and cathether related complications
- gastric lap band catheter malfunction
- port-catheter disconnection
- catheter-band disconnection
- proximal oesophageal dilatation without stomal stenosis
- oesophageal dysmotility and reflux / oesophageal gastrification
Fluoroscopy is a versatile tool in assessing for potential gastric band complications. It can not only demonstrate most complications, but also supports percutaneous adjustment of band inflation and dynamic stoma assessment.
CT has the advantage of demonstrating other causes of abdominal pain in these patients and is increasingly used in their acute assessment. This is usually performed with the administration of 15-20 mL water soluble oral contrast agent prior to assessment.
This is usually evident on plain radiographs with an abnormal Phi angle and band lie. The band can be misplaced into pergastric fat or distal stomach with complicating outlet obstruction.
This is rare, reported in < 1% of patients. On fluoroscopy it is evident as contrast tracking outside the gastric outline.
On CT there is extra-luminal contrast, penetration of the gastric wall by the band, localized gas locules or pneumoperitoneum.
This is seen as concentration dilation of the gastric stoma and results from stomal stenosis, adhesions or nutritional overload. Findings include a tight stoma with delayed gastric emptying. Axial herniation of the stomach, oseophageal reflux and dilation can occur as late complication.
This can occur with either anterior or posterior upward herniation of the distal stomach upwards through the band. There is usually resultant eccentric as opposed to concentric pouch dilation.
Fluoroscopic examination can be normal in the early stages of intragastric erosion. Later on oral contrast is seen pooling around the band tubing outside of the gastric lumen.
CT may show eccentric gastric thickening with invasion of the band into the gastric lumen. Sometimes locules of gas are seen within the gastric wall underlying the band.
Oesophageal dilation or dysmotility
This is demonstrated fluoroscopically. Secondary complication from dysmotility or dilation such a gastro-oesophgeal reflux, oesophagitis and aspiration may also be demonstrated.
Connector tube or port complications
Tube disconnection can be visible on radiographs as discontinuity in the tube catheter. This is usually at the junction with the port or band. Fluoroscopic examination with the injection of 5 mL of contrast into the tube port can show a contrast blush to confirm the site of leakage.
Port-site infection which is not evident externally can be assessed with ultrasound to look for abscess formation.
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