Osteochondritis dissecans - stage III

Case contributed by Andrei Tsoriev , 5 May 2016
Diagnosis certain
Changed by Henry Knipe, 6 May 2016

Updates to Study Attributes

Findings was changed:

Intraarticular fragment best seen on T1 sagittal images due to its calcification and also on axial plane.

Medial femoral condyle deformation with a cartilage interruption.

No evidence of ligamentous and/or meniscal damagesinjury. 

Normal patella and trochlear anatomy.

Sesamoid bone posterior to the lateral femoral condyle - fabella.

Updates to Quizquestion Attributes

Question was changed:
What is a pickthe peak age for this disease?

Updates to Quizquestion Attributes

Question was changed:
WhichWhat is the eponymeponymous name of this disease?
Answer was changed:
Konig disease.

Updates to Quizquestion Attributes

Answer was changed:
In accordance towith the osteochondritis dissecans surgical staging system - stage III.
Question was changed:
What stage of disease is this conditioncase?

Updates to Case Attributes

Body was changed:

ThereThese are findings of an osteochondritis dissecans (Konig disease) outcome - formed intraarticularwith an intra-articular non dislocated-dislocated fragment, in accordance to the osteochondritis dissecans surgical staging system stage III.

There were specific complaints to suspect this pathology: pains pain increasing with knee joint motion, which is thought to be caused by intraarticularintra-articular fragment impact on toimpaction onto well innervated knee joint structures.

Osteochondritis dissecans occurs most often in children and adolescents, and most commonly in the knee joint, but also can beebe seen in the elbows, and ankles, and other joints such aas the talus. Estimated prevalence in the knee is 15 to 29 per 100,000 individuals.

The exact cause of osteochondritis dissecans is unknown. The reduced blood flow to the epiphysis of the affected bone might result from repetitive trauma — small, multiple episodes of minor, unrecognised injury that damage the bone. There might be a genetic component, making some people more prone to the disorder. There are some ongoing trials conserningconcerning genetic changes. 

  • -<p>There are findings of an osteochondritis dissecans (Konig disease) outcome - formed intraarticular non dislocated fragment, in accordance to the osteochondritis dissecans surgical staging system stage III.</p><p>There were specific complaints to suspect this pathology: pains increasing with knee joint motion, which is thought to be caused by intraarticular fragment impact on to well innervated knee joint structures.</p><p>Osteochondritis dissecans occurs most often in children and adolescents and most commonly in the knee joint, but also can bee seen in elbows, ankles and other joints such a talus. Estimated prevalence in knee is 15 to 29 per 100,000 individuals.</p><p>The exact cause of osteochondritis dissecans is unknown. The reduced blood flow to the epiphysis of the affected bone might result from repetitive trauma — small, multiple episodes of minor, unrecognised injury that damage the bone. There might be a genetic component, making some people more prone to the disorder. There are some ongoing trials conserning genetic changes. </p><p> </p><p> </p><p> </p>
  • +<p>These are findings of osteochondritis dissecans (Konig disease) - with an intra-articular non-dislocated fragment, in accordance to the osteochondritis dissecans surgical staging system stage III.</p><p>There were specific complaints to suspect this pathology: pain increasing with knee joint motion, which is thought to be caused by intra-articular fragment impaction onto well innervated knee joint structures.</p><p>Osteochondritis dissecans occurs most often in children and adolescents, and most commonly in the knee joint, but also can be seen in the elbows and ankles, and other joints such as the talus. Estimated prevalence in the knee is 15 to 29 per 100,000 individuals.</p><p>The exact cause of osteochondritis dissecans is unknown. The reduced blood flow to the epiphysis of the affected bone might result from repetitive trauma — small, multiple episodes of minor, unrecognised injury that damage the bone. There might be a genetic component, making some people more prone to the disorder. There are some ongoing trials concerning genetic changes. </p>

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