Classic clinical triad in tuberous sclerosis:
Seizures, intellectual disability, adenoma sebaceum
Extracranial findings in tuberous sclerosis:
Cardiac rhabdomyomas, renal (and less commonly hepatic) AMLs, renal cysts, oncocytomas, LAM/LAM-like cystic lung changes
An enhancing mixed solid/cystic lesion contacts the lateral wall of the right lateral ventricle and the right foramen of Monro. Assymetric enlargement of the right lateral ventricle with leftward bowing of the left lateral ventricle.
A smaller non-enhancing nodular lesion arises from the posterior body of the left ventricle/left trigone.
There are multiple cortical and subcortical regions of FLAIR hyperintensity without enhancement, reflecting cortical tubers. Areas of low T1 and T2 are likely to represent calcified tubers.