Acute right MCA infarct, meningioma and retinal detachment (CT perfusion)
Acute left hemiplegia. AF, INR 3.9. ? hemorrhage.
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Right frontal lobe encephalomalacia with confluent white matter hypoattenuation involving the right frontal and right parietal lobe unchanged from the prior study 6 moths earlier consistent with an old right MCA infarct.
Hyperdense right MCA consistent with acute thrombus.
Hyperattenuating extra axial mass arising from the right sphenoid ridge measuring 2.5cm. Atrophic changes with prominence of the cortical sulci and ventricular system. Brainstem and cerebellum are within normal limits. No acute intraparenchymal hemorrhage.
Unchanged hypoattenuation within the right lobe most consistent with retinal detachment with hemorrhage.
Calcification of the aortic arch. Origin of the innominate artery is not identified. The common carotids, internal carotids, vertebral and basilar arteries are patent. Calcified atheroma of the right and left carotid bulb with no haemodynamically significant stenosis. Tortuosity of the right internal carotid artery.
Bilateral carotid siphon calcification. Normal circle of Willis anatomy. Occlusive thrombus within the proximal right MCA (M1). Normal contrast opacification of the anterior cerebral, posterior cerebral and left middle cerebral arteries.
Perfusion imaging is confounded by the presence of the previous right MCA infarct. Reduced CBF within the right frontal lobe consistent with a region of established chronic infarct present on the non-contrast CT. MTT and Tmax are increased within the entire right MCA territory consistent with proximal occlusive thrombus, although the volume of the acute perfusion deficit is difficult to establish given the prior right MCA infarct.
- Occlusive thrombus of the proximal right MCA.
- Old established right MCA infarct unchanged from previous CT 6 months earlier.
- Right-sided meningioma centered in the right sphenoid ridge.
- Right retinal detachment.
The right M1 thrombus was removed with endovascular clot retrieval 4 hours post onset of hemiplegia.
Incidental retinal hemorrhage was secondary to the anticoagulation.