Anoxic brain injury secondary to cardiac arrest
Post cardiac arrest.
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Evidence of hypoxic-ischemic brain injury. Widespread loss of grey-white matter differentiation.
Reversal sign is present within the posterior cerebellum. Pseudo-subarachnoid hemorrhage is seen in the hyper-dense appearance of the falx cerebri and tentorium cerebelli compared to the surrounding edematous brain tissue.
Extensive anoxic brain injury secondary to cardiac arrest.
Hypoxic-ischemic brain injury is a common sequela of prolonged cardiac arrest, and can present clinically ranging from subtle cognitive impairment to brain death1.
Other causes of hypoxic-ischemic brain injury in adults include hypoxia secondary to severe respiratory failure, asphyxiation, drowning, and carbon monoxide poisoning.
Case submitted by Dr. N. Larocque.