Presentation
Normotensive patient with right sided weakness and disturbed level of consciousness.
Patient Data
Multiple left occipital, as well as parietal and high posterior frontal hemorrhagic mass lesions, are seen with the largest being about 3.5 X 2.5 cm. They demonstrate predominately hyperintense T1 and T2 signals with iso- to hypointense core. The lesions show blooming in GRE sequence. They show no evident post-contrast enhancement. They are surrounded by perifocal vasogenic brain edema with some compression of the occipital horn and trigone of the left lateral ventricle. A tiny few millimetric lesion with GRE blooming is seen in the right parietal lobe.
Mild dilatation of the ventricular system with patchy T2/FLAIR hyperintense foci at the periventricular and deep white matter.
Widened extra-axial CSF spaces; no extra-axial collection.
Normal sellar region and posterior fossa.
Mild atherosclerotic changes of the cerebral arteries with mild irregularity. Otherwise normal signal and caliber of the cerebral arteries with no occluded segment or significant stenosis.
No aneurysmal dilatation or A-V malformation.
Attenuated right vertebral artery, otherwise, normal vertebrobasilar system.
Case Discussion
Bilateral; predominately left cerebral subacute hemorrhagic, non-enhancing mass lesions; these lobar hemorrhages are suggestive of amyloid angiopathy; lack of enhancement makes hemorrhagic metastatic deposits much less likely possibility. Consider amyloid angiopathy in normotensive elderly patients with multifocal non-enhancing lobar hemorrhage.
Atrophic brain changes with periventricular arteriosclerotic leucoencephalopathy. Mild atherosclerotic changes of the cerebral arteries, with no other significant abnormality.
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