Left parietal lobe and volume loss, T2 FLAIR white matter hyperintensity, and gyriform haemosiderin staining representing site of resolving intraparenchymal haematoma. Diffuse cerebral and cerebellar volume loss within normal limits for patient of this age. Confluent periventricular white matter and subcortical T2 and FLAIR hyperintensity in keeping with chronic small vessel disease.
Additionally, susceptibility weighted sequences demonstrate multiple punctate foci of blooming in both cerebral hemispheres (with predominance at the grey-white matter interface), brainstem and right cerebral hemisphere.
Conclusion: Diffuse punctate foci of blooming artefact on susceptibility weighted sequences is highly suggestive of cerebral amyloid angiopathy.