Presentation
Trauma. No seizure, weakness, or tremor. Normal neurological examination.
Patient Data
Age: 35 years
Gender: Male
From the case:
Fahr disease
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Info
Diffuse symmetrical calcifications in the bilateral basal ganglia, thalami, midbrain (including red nuclei), pons, cerebellar hemispheres (including dentate nuclei), and subcortical white matter of the fronto-parieto-temporal lobes.
Case Discussion
Primary Fahr disease should only be considered when secondary causes of a Fahr syndrome are excluded, such as pseudohypoparathyroidism, mitochondrial diseases, infections (e.g. CMV, HIV), and radiation exposure.
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