Inferior vena cava web

Case contributed by Abdallah Al Khateeb , 17 Jan 2019
Diagnosis almost certain
Changed by Abdallah Al Khateeb, 5 Jun 2022

Updates to Case Attributes

Body was changed:

The patient had abdominal pain, ascites and hepatomegaly: a triad classically seen with Budd-Chiari syndrome (BCS) or hepatic venous outflow obstruction. BCS has many aetiologies, and this CT directly shows one of the uncommon causes: inferior vena cava web.

An IVC web can be congenital, or more commonly, a sequel of prior thrombosis.

This patient underwent inferior venacavography (images not available) which confirmed an IVC web, and angioplasty was performed thereafter.

The liver parenchymal nodules, volume redistribution (e.g. caudate hypertrophy), and features of portal hypertension (e.g. splenomegaly, collaterals), are typically seen with chronic BCS.

Although the hepatic nodules are likely regenarative, hepatocellular carcinoma remains a concern, and cannot be reliably ruled out on a monophasic study. The ideal imaging evaluation of such a case must include liver protocol CT or MRI.

  • -<p>The patient had abdominal pain, ascites and hepatomegaly: a triad classically seen with <a href="/articles/budd-chiari-syndrome-1">Budd-Chiari syndrome</a> (BCS) or hepatic venous outflow obstruction. BCS has many aetiologies, and this CT directly shows one of the uncommon causes: <a href="/articles/inferior-vena-cava-web">inferior vena cava web</a>.</p><p>An IVC web can be congenital, or more commonly, a sequel of prior thrombosis.</p><p>This patient underwent inferior venacavography (images not available) which confirmed an IVC web, and angioplasty was performed thereafter.</p><p>The liver parenchymal nodules, volume redistribution (e.g. caudate hypertrophy), and features of portal hypertension (e.g. splenomegaly, collaterals), are typically seen with chronic BCS.</p><p>Although the hepatic nodules are likely regenarative, hepatocellular carcinoma remains a concern, and cannot be reliably ruled out on a monophasic study. The ideal imaging evaluation of such a case must include liver protocol CT or MRI.</p>
  • +<p>The patient had abdominal pain, ascites and hepatomegaly: a triad classically seen with <a href="/articles/budd-chiari-syndrome-1">Budd-Chiari syndrome</a> (BCS) or hepatic venous outflow obstruction. BCS has many aetiologies, and this CT directly shows one of the uncommon causes: <a href="/articles/inferior-vena-cava-web">inferior vena cava web</a>.</p><p>An IVC web can be congenital, or more commonly, a sequel of prior thrombosis.</p><p>This patient underwent inferior venacavography (images not available) which confirmed an IVC web, and angioplasty was performed thereafter.</p><p>The liver parenchymal nodules, volume redistribution (e.g. caudate hypertrophy), and features of portal hypertension (e.g. splenomegaly, collaterals), are typically seen with chronic BCS.</p><p>Although the hepatic nodules are likely <a title="regenarative" href="/articles/regenerative-liver-nodule">regenarative</a>, hepatocellular carcinoma remains a concern, and cannot be reliably ruled out on a monophasic study. The ideal imaging evaluation of such a case must include liver protocol CT or MRI.</p>

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