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No intra- or extra-axial collection or haemorrhage demonstrated. Hyperdense basilar tip and left P1/2 segments. Loss of grey-white differentiation in the left PCA territory.
Perfusion imaging shows a small infarct core in the left PCA territory with larger area of ischaemic penumbra.
Filling defect within the very distal basilar artery extending into the left P1/2 segments. Opacification of the left P3/4 segments via left PComA. Both PICAs, AICAs, SCAs and right PCA are patent.