Myelin oligodendrocyte glycoprotein antibody-associated disease (MOGAD)
Presentation
Fever with sudden diminished vision.
Patient Data
Multiple asymmetric cortical / subcortical white matter T2/FLAIR hyperintense lesions are seen in bilateral cerebral hemispheres extending to corpus callosum at places. Subtle hyperintensities are seen in bilateral medial thalami.
Thickening and hyperintensity of intraocular, intraorbital segments of both optic nerves is seen with sparing of intracanalicular, intracranial segments and optic chiasma, suggesting bilateral optic neuritis.
Laboratory investigations turned out to be positive for serum anti-MOG antibodies.
Case Discussion
Lack of involvement of optic chiasma, tracts and area postrema with history of fever favors the diagnosis of Myelin oligodendrocyte glycoprotein antibody-associated disease (MOGAD). Laboratory investigations turned out to be positive for serum anti-MOG antibodies.
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