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No acute intra- or extra-axial haemorrhage demonstrated. Periventricular and deep white matter hypoattenuation, most in keeping with chronic small vessel ischaemic change. Old left cerebellar hemisphere infarct. Hypodensities within both basal ganglia / internal capsules either represent chronic lacunar infarcts or prominent perivascular spaces. Grey-white differentiation is elsewhere preserved. Increased density of the left M1 segment at its bifurcation. Ventricular size, sulcal pattern and basal cisterns all appear age-appropriate.