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The patient had history of hydrocephalus and unerwent VP shunt
Complex right temporal intra axial lesion. In T1 it displays heterogenous intensity predominantly low signal in the center of the lesion with peripheral hyperintensity. In T2 the lesion is predominantly hyperintense in the center and periphery. The peripheral component becomes low signal in Fat suppressed T2. Scattered foci of hyperintense signal in T1 and FLAIR are seen dessiminated within the subarachnoid space.