Subfalcine herniation from complete MCA infarct, complicated by ACA infarct
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An ACA territory infarct can occur as a complication of an MCA territory infarct - midline shift from vasogenic oedema results in stretching and eventual occlusion of the anterior cerebral artery.
-<p><a title="anterior cerebral artery territory infarct" href="/articles/anterior-cerebral-artery-territory-infarct">ACA territory infarct</a> can occur as a complication of <a title="MCA territory infarcts" href="/articles/middle-cerebral-artery-infarction">MCA territory infarct</a> - <a title="Midline shift" href="/articles/midline-shift">midline shift</a> from vasogenic oedema results in stretching and eventual occlusion of the anterior cerebral artery. </p>- +<p>An <a href="/articles/anterior-cerebral-artery-aca-infarct">ACA territory infarct</a> can occur as a complication of an <a href="/articles/middle-cerebral-artery-mca-infarct">MCA territory infarct</a> - <a href="/articles/midline-shift">midline shift</a> from <a title="Vasogenic oedema" href="/articles/vasogenic-cerebral-oedema">vasogenic oedema</a> results in stretching and eventual occlusion of the anterior cerebral artery.</p>
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Complete right MCA territory infarct with pronounced midline shift (subfalcine(subfalcine herniation). The right thalamus is displaced to the left of midline (not shown). Ipsilateral distal ACA is likely to be complicatedcompressed by the subfalcine herniation with, resulting in infraction of the right superior frontal gyrus.
The right lateral ventricle is effaced by the mass effect with a "trapped" left lateral ventricle. Periventricular hypodensities at the trigone region of the left lateral ventricle may represent transepenedymal migration of CSF.
Apparent hyperdensities along the region of Sylvian fissure, cortical sulci and M1 segment of MCA may reflect increaseincreased cerebral pressure - the so called "pseudosubarachnoid sign-called "pseudosubarachnoid haemorrhage". Alternatively, the hyperdense M1 may represent insitu an in-situ thrombus.