The diffuse periventricular white matter T1 hyperintensity and restricted diffusion identified
previously have evolved further. There is now extensive cystic necrosis in these areas. This is manifest as diffuse high T2 signal and low T1 signal with reversal of the previous restricted diffusion/
The process is limited to the white matter with complete sparing of the overlying cortex.
No evidence of encephalomalacia or atrophy at present.
Minor areas of gliosis are present at the margins of the cystic necrosis bilaterally.
The sparing of the cortex and basal ganglia suggest a toxic aetiology rather than hypoxia.