Risk factors for cerebral venous thrombosis:
Pregnancy, OCP, malignancy, sepsis, prothrombotic haematological conditions
Vein involved in: bilateral thalami infarcts, parasagittal frontal/parietal lobe infarcts
Bilateral thalami - internal cerebral veins (also think artery of Percheron); bilateral parasagittal frontal/parietal lobes - superior sagittal sinus
The left vein of Trolard is non-opacified through its course. A 2 cm non-occlusive filling defect is present within the superior sagittal sinus.
An ovoid area of T2 and FLAIR hyperintensity is present within the left anterior parietal and posterior frontal lobe. This region demonstrates large areas of diffusion restriction, with associated susceptibility artefact on SWI, in keeping with intraparenchymal haemorrhage.
The area of diffusion restriction and SWI artefact involves the post-central gyrus laterally. T2 and FLAIR hyperintensity, in keeping with oedema, extends into the precentral gyrus.
Mild mass effect is exerted on the adjacent parenchyma, with effacement of the superior parietal sulci.
Thin, continuous cortical enhancement is evident.
No significant rightward midline shift.