Altered signal intensity lesions in bilateral parieto - occipital periventricular region and splenium/posterior body of corpus callosum, with associated ependymal thickening and peri lesional edema ; and displaying Iso- hypointense signals on T1 WI getting Iso- hyperintense on T2WI with areas on diffusion restriction in splenium of corpus callosum. No areas of GRE blooming to suggest bleed.
On CEMR, the lesions show heterogeneous nodular ependymal enhancement in posterior periventricular and splenium/posterior body of corpus callosum regions.