Hypoattenuation with loss of grey-white matter differentiation through the right inferior cerebellar hemisphere. Ill-defined cortically based hyperattenuation posteriorly. With contrast there is no definite enhancement in this region, and no other enhancing intracranial foci.
Significant local mass effect with deviation of the falx cerebelli 8mm to the left. Descent of the right cerebellar tonsil down to the level of the foramen magnum.
Lateral ventricles remain patent with hydrocephalus or midline shift.
- Probable large subacute infarct of the right inferior cerebellar hemisphere, corresponding to a right PICA vascular territory.
- Right posterior cerebellar cortical hyperattenuation could relate to early haemorrhagic transformation or cortical laminar necrosis.