Neuropsychology testing demonstrated profound impairment in non-verbal skills, profound slowing on non-verbal tasks ad marked impairment in aspects of verbal skills. Her memory was inefficient and slowed, with no rapid forgetting. Her performance on executive tasks was colored by her visuospatial impairment and processing speed. These findings are consistent with posterior cortical atrophy.

CSF demonstrated decreased levels of Abeta1-42, which is consistent with Alzheimer's Disease, as this protein is deposited and aggregated in amyloid plaques and hence decreased.