Findings:
Asymmetrical hyperdense left MCA, A1 ACA and left terminal ICA involving the supraclinoid, ophthalmic and cavernous segment suggestive of large vessel thromboembolism.
Grey-white matter differentiation loss and generalised hypoattenuation of the left ACA and MCA vascular territory. Cytotoxic oedema secondary to established ACA and MCA territory infarct.
No macroscopic haemorrhagic transformation.
The left cerebral uncal process overhangs the left falx cerebelli concerning for early uncal herniation.
Summary:
Established left MCA and ACA vascular territory cerebral infarction with cytotoxic oedema, mass effect and early left uncal herniation.
Hyperdense left intracranial ICA, MCA (to mid M1) and ACA A1 segment suggestive of acute thromboembolism.
No haemorrhagic transformation.