Multiple ill-defined small arterial hyper-enhancing foci throughout the enlarged liver. Early opacification of the intrahepatic portal vein branches in the arterial phase favors arterioportal shunting. Evidence of cirrhosis and portal hypertension are visible as mild liver surface irregularity, ascites, splenomegaly, and small omental collaterals. The common hepatic artery is hypertrophied and the aorta becomes small below the celiac trunk.