Chronic occlusion of the right coronary artery

Case contributed by Joachim Feger
Diagnosis certain

Presentation

Atypical chest pain. History of smoking and hyperlipidemia.

Patient Data

Age: 45 years
Gender: Male
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Axial
non-contrast
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Axial C+
arterial phase
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Short axis C+
arterial phase
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VLA C+
arterial phase
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Curved
RCA
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Curved
LAD
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Curved
CX
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Curved
OM1
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3D
Heart
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3D
Coronaries
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Info

Technique

  • patient premedication: beta blocker and nitrates

  • acquisition method: step and shoot (prospective acquisition - 100 KV)

  • contrast injection protocol: triphasic injection

  • image reconstruction: standard without edge correction

Findings

Plaque burden:

  • calcium score (according to Agatson, not shown): 104

  • segment involvement score: 3-4

Right coronary artery (RCA): double distal segment

Left main: inconspicuous

Left anterior descending artery (LAD): two diagonal branches

  • eccentric mixed plaque in the proximal segment without significant stenosis

  • thin, rudimentary first diagonal branch

  • strong second diagonal branch without stenosis

  • stair step artifact in the distal segment

Circumflex artery (CX): two marginal branches (OM1 & OM2) and posterolateral branch

  • eccentric calcified plaque of the proximal segment

  • thin first marginal branch with at least moderate stenosis proximally (D: 50-69%)

  • no plaques or stenosis of the second marginal and posterolateral branches

  • stair step artifact in the distal segment

Impression

  • chronic occlusion of the right coronary with distal collateralisation

  • proximal moderate stenosis of a thin first marginal branch

  • moderate amount of coronary plaque

  • CAD-RADS 5/P2

  • massive bilateral hilar and mediastinal lymphadenopathy

Exam courtesy: Silva Reinecke (medical imaging technologist)

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Axial C+ portal
venous phase
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Coronal C+ portal
venous phase
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Axial lung
window
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Coronal
lung window
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Massive bilateral hilar and mediastinal lymphadenopathy.

No lung mass was detected.

Mild emphysema.

Cardiac MRI

mri
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Axial
SSFP
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2ch Cine
SSFP
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4ch Cine
SSFP
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3ch Cine
SSFP
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Short axis
Cine SSFP
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Short
axis STIR
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T1 mapping
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Short axis
Perfusion - stress
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Short axis Cine
SSFP - stress
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Short axis
Perfusion - rest
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2ch
IRGE
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4ch
IRGE
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3ch
IRGE
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Short
axis IRGE
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Heart rate: ~70 bpm at rest -> 90 bpm during stress perfusion after Adenosin 140 µg/kg/min

Image quality: mild to moderate respiratory artifacts, otherwise no limitations

Morphology and functional analysis (endo-volume):

  • LV-EDVI: 75 mL/m²

  • LV-ESVI: 38 mL/m²

  • LV-SVI: 37 mL/m²

  • LV-EF: 49%

  • cardiac output: 5.8 L/min

  • cardiac index: 2.6 L/min/m²

  • LV-ED wall mas index (without papillary muscles): 67 g/cm

  • septum and inferior wall thickness: 9 and 8 mm

Findings:

Mild hypokinesia in basal inferoseptal and inferior segments.

No evidence of focal myocardial edema on STIR.

Mildly elevated T1 mapping values in septal and inferior segments (z-score: 2-3).

Small subendocardial perfusion defect under Adenosin stress in inferoseptal and inferior midventricular segments not visible during rest.

No intracavitary thrombi.

Impression:

Cardiac MRI findings are consistent with mild myocardial ischemia in midventricular inferioseptal and inferior segments.

Norma viability of the myocardium. No signs of infarction or myocardial scar tissue.

Mildly elevated T1 mapping values possibly mild myocardial edema.

Exam courtesy: Jeanette Moses & Tobias Jahn (medical imaging technologists)

Case Discussion

A case of a patient with chronic occlusion of the right coronary artery with mild myocardial ischemia and partial collateralisation via branches of the circumflex artery. Myocardial viability is preserved with no signs of myocardial infarction or myocardial scar tissue.

The RCA occlusion was confirmed by cardiac catheterization, which also showed the collateral pathway via branches from the circumflex artery. The stenosis of OM1 was not significant.

Based on these findings, the patient is very likely to benefit from revascularization 1 and was therefore transferred to a cardiac center.

The bilateral hilar and mediastinal lymphadenopathy is further investigated oncologically.

Courtesy: Dr Soeren Linsel (cardiologist)

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