Fahr disease: juvenile onset
Progressive deterioration of cognitive and motor functions.
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Bilaterally symmetrical hyperdense (HU + 278) lesions, suggestive of calcification, involving globus pallidus, putamen, caudate nucleus, internal capsule, thalami, dentate nucleus, cerebellum and subcortical white matter.
Blood chemistry revealed normal serum levels of calcium, phosphorus and alkaline phosphatase.
The CT scan findings when correlated with typical clinical history and normal blood chemistry were suggestive of Fahr disease.