Presentation
Fall. Parapelgia.
Patient Data
Normal craniocervical junction.
C5/6- Large disc prolapse at C5/C6. This combined with posterior longitudinal ligament calcification is severely compressing and distorting the cord, with both high signal and expansion of the cord at the C5/C6 level, consistent with the cord compression with cord edema opposite the C5 and C6 vertebral bodies. Minimal calcification within the prolapsed disc suggesting a large component of this is acute.
Bilateral foraminal stenosis also at C5/C6 due to neurocentral osteophytosis.
Small left paracentral disc osteophyte complex at T6-T7. No cord compression or nerve root impingement.
Left paracentral disc osteophyte complex at T10/T11 contacting the ventral aspect of the cord with mild exit foraminal narrowing. No nerve root impingement.
Diffuse disc osteophyte complex T11/T12 with left-sided asymmetry, contacting the ventral aspect of the cord. No cord or nerve root impingement.
Case Discussion
Traumatic C5/C6 cord compression due to an acute disc prolapse on the background of chronic disc disease.
CT was undertaken prior to the MRI on which no bony injury was identified.
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