Valgus extension overload syndrome

Changed by Amir Rezaee, 6 Jan 2019

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Valgus extension overload syndrome is referred to a constellation of symptoms and pathologies commonly seen in over head throwing athletes secondary to high repetitive tensile, shear and compressive forces generated by the overhead throwing motion. The hallmark of valgus extension overload syndrome is injury to ulnar collateral ligament.

Epidemiology

Mostly over head throwing young athletes such as baseball players or javelin throwers.

Clinical presentation

Typical history is an athlete with posterior elbow pain at ball release. Pain reported in posteromedial elbow at full extension or during either acceleration or deceleration phases of pitch. Restriction in full extension is common in throwing athletes in their dominant elbow, however this finding can be more pronounced in this entity.

On physical examination, focal tenderness to palpation over posteromedial elbow join and crepitus are common.  

Symptoms can be reproduced  by placing a valgus stress on the elbow at 20 to 30 degrees of flexion while forcing the elbow into terminal extension.

Pathology

Extreme tensile stress forces applies to the anterior band of the ulnar collateral ligament during forced extension due to high valgus torque. This can injure ulnar collateral tendon either in acute setting or chronically due to micro trauma.  

Other excessive forces during pitch are high shear stresses in posterior compartment causing cartilage injury of the olecranon and compressive forces radiocapitular joint with resultant osteonecrosis and AVN particularly in skeletally immature athletes.  

Radiographic features

On X-ray chronic changes can be detected such as osteophyte formation in the posteromedial olecranon fossa, loose bodies, capitellum osteochondral defects, calcium deposits or ossification of the ulnar collateral ligament.

MRI is the modality of choice for imaging assessment of these patients:

Complete UCL tear is diagnosed by ligament discontinuity and abnormal fiber laxity. Partial thickness tears of UCL are most commonly occur in mid substance and characterized by peri ligamentous edema and varying degrees of fiber discontinuity and increased signal intensity on fluid sensitive images traversing the ligament fibers. Fuid or contrast material insinuating below the ligament along the margin of the bone at the sublime tubercle known as the T sign can be observed due to partial avulsion of the UCL and periosteal stripping.

Treatment

Initially non invasive treatments such as NSAIDS,activity modification, steroid injections, and rehabilitation attempted. Complete tears and failed conservative treatments after 3 months particularly in highly active athletes may indicate surgical UCL reconstruction with graft, known as  "Tommy John surgery".

  • -<p><strong>Valgus extension overload syndrome </strong>is referred to a constellation of symptoms and pathologies commonly seen in over head throwing athletes secondary to high repetitive tensile, shear and compressive forces generated by the overhead throwing motion. The hallmark of valgus extension overload syndrome is injury to <a href="/articles/medial-collateral-ligament-complex-of-the-elbow">ulnar collateral ligament</a>.</p><h4>Epidemiology</h4><p>Mostly young athletes.</p><h4>Clinical presentation</h4><p>Typical history is an athlete with posterior elbow pain at ball release. Pain reported in posteromedial elbow at full extension or during either acceleration or deceleration phases of pitch. Restriction in full extension is common in throwing athletes in their dominant elbow, however this finding can be more pronounced in this entity.</p><p>On physical examination, focal tenderness to palpation over posteromedial elbow join and crepitus are common.  </p><p>Symptoms can be reproduced  by placing a valgus stress on the elbow at 20 to 30 degrees of flexion while forcing the elbow into terminal extension.</p><h4>Pathology</h4><p>Extreme tensile stress forces applies to the anterior band of the ulnar collateral ligament during forced extension due to high valgus torque. This can injure ulnar collateral tendon either in acute setting or chronically due to micro trauma.  </p><p>Other excessive forces during pitch are high shear stresses in posterior compartment causing cartilage injury of the olecranon and compressive forces radiocapitular joint with resultant osteonecrosis and AVN particularly in skeletally immature athletes.  </p><h4>Radiographic features</h4><p> </p><p>MRI is the modality of choice for imaging assessment of these patients:</p><p>Complete UCL tear is diagnosed by ligament discontinuity and abnormal fiber laxity. Partial thickness tears of UCL are most commonly occur in mid substance and characterized by peri ligamentous edema and varying degrees of fiber discontinuity and increased signal intensity on fluid sensitive images traversing the ligament fibers. Fuid or contrast material insinuating below the ligament along the margin of the bone at the sublime tubercle known as the T sign can be observed due to partial avulsion of the UCL and periosteal stripping.</p>
  • +<p><strong>Valgus extension overload syndrome </strong>is referred to a constellation of symptoms and pathologies commonly seen in over head throwing athletes secondary to high repetitive tensile, shear and compressive forces generated by the overhead throwing motion. The hallmark of valgus extension overload syndrome is injury to <a href="/articles/medial-collateral-ligament-complex-of-the-elbow">ulnar collateral ligament</a>.</p><h4>Epidemiology</h4><p>Mostly over head throwing young athletes such as baseball players or javelin throwers.</p><h4>Clinical presentation</h4><p>Typical history is an athlete with posterior elbow pain at ball release. Pain reported in posteromedial elbow at full extension or during either acceleration or deceleration phases of pitch. Restriction in full extension is common in throwing athletes in their dominant elbow, however this finding can be more pronounced in this entity.</p><p>On physical examination, focal tenderness to palpation over posteromedial elbow join and crepitus are common.  </p><p>Symptoms can be reproduced  by placing a valgus stress on the elbow at 20 to 30 degrees of flexion while forcing the elbow into terminal extension.</p><h4>Pathology</h4><p>Extreme tensile stress forces applies to the anterior band of the ulnar collateral ligament during forced extension due to high valgus torque. This can injure ulnar collateral tendon either in acute setting or chronically due to micro trauma.  </p><p>Other excessive forces during pitch are high shear stresses in posterior compartment causing cartilage injury of the olecranon and compressive forces radiocapitular joint with resultant osteonecrosis and AVN particularly in skeletally immature athletes.  </p><h4>Radiographic features</h4><p>On X-ray chronic changes can be detected such as osteophyte formation in the posteromedial olecranon fossa, loose bodies, capitellum osteochondral defects, calcium deposits or ossification of the ulnar collateral ligament.</p><p>MRI is the modality of choice for imaging assessment of these patients:</p><p>Complete UCL tear is diagnosed by ligament discontinuity and abnormal fiber laxity. Partial thickness tears of UCL are most commonly occur in mid substance and characterized by peri ligamentous edema and varying degrees of fiber discontinuity and increased signal intensity on fluid sensitive images traversing the ligament fibers. Fuid or contrast material insinuating below the ligament along the margin of the bone at the sublime tubercle known as the T sign can be observed due to partial avulsion of the UCL and periosteal stripping.</p><h4>Treatment</h4><p>Initially non invasive treatments such as NSAIDS,activity modification, steroid injections, and rehabilitation attempted. Complete tears and failed conservative treatments after 3 months particularly in highly active athletes may indicate surgical UCL reconstruction with graft, known as  "Tommy John surgery".</p>

References changed:

  • 1. Dugas JR. Valgus extension overload: diagnosis and treatment. (2010) Clinics in sports medicine. 29 (4): 645-54. <a href="https://doi.org/10.1016/j.csm.2010.07.001">doi:10.1016/j.csm.2010.07.001</a> - <a href="https://www.ncbi.nlm.nih.gov/pubmed/20883902">Pubmed</a> <span class="ref_v4"></span>
  • 2. Bucknor MD, Stevens KJ, Steinbach LS. Elbow Imaging in Sport: Sports Imaging Series. (2016) Radiology. 279 (1): 12-28. <a href="https://doi.org/10.1148/radiol.2016150501">doi:10.1148/radiol.2016150501</a> - <a href="https://www.ncbi.nlm.nih.gov/pubmed/26989928">Pubmed</a> <span class="ref_v4"></span>
  • 3. Ahmad CS, ElAttrache NS. Valgus extension overload syndrome and stress injury of the olecranon. (2004) Clinics in sports medicine. 23 (4): 665-76, x. <a href="https://doi.org/10.1016/j.csm.2004.04.013">doi:10.1016/j.csm.2004.04.013</a> - <a href="https://www.ncbi.nlm.nih.gov/pubmed/15474228">Pubmed</a> <span class="ref_v4"></span>

Systems changed:

  • Musculoskeletal

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