Brain cortical laminar necrosis
Diagnosis almost certain
Disclosures
- updated 6 Apr 2022:
- Radiopaedia Events Pty Ltd, Speaker fees (past)
- Integral Diagnostics, Shareholder (ongoing)
- Micro-X Ltd, Shareholder (ongoing)
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Cardiac arrest maybe is the most characteristic example of causes of hypoxic-ischemic-ischaemic brain damage.
The vulnerability of cortical layers 3, 4, and 5 to anoxia and ischemiaischaemia, due to its higher metabolic demands, is the cause of cortical laminar necrosis.
The gyriform high attenuation is believed to be caused by the accumulation of denatured proteins in dying cells and does not represent the presence of hemorrhagehaemorrhage.
-<p>Cardiac arrest maybe is the most characteristic example of causes of <a href="/articles/hypoxic-ischaemic-encephalopathy-adults-and-children">hypoxic-ischemic brain damage</a>.</p><p>The vulnerability of cortical layers 3, 4, and 5 to anoxia and ischemia, due to its higher metabolic demands, is the cause of <a href="/articles/cortical-laminar-necrosis">cortical laminar necrosis</a>. </p><p>The gyriform high attenuation is believed to be caused by the accumulation of denatured proteins in dying cells and does not represent the presence of hemorrhage.</p>- +<p>Cardiac arrest maybe is the most characteristic example of causes of <a href="/articles/hypoxic-ischaemic-encephalopathy-adults-and-children">hypoxic-ischaemic brain damage</a>.</p><p>The vulnerability of cortical layers 3, 4, and 5 to anoxia and ischaemia, due to its higher metabolic demands, is the cause of <a href="/articles/cortical-laminar-necrosis">cortical laminar necrosis</a>. </p><p>The gyriform high attenuation is believed to be caused by the accumulation of denatured proteins in dying cells and does not represent the presence of haemorrhage.</p>
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