Fahr syndrome
Diagnosis almost certain
Updates to Case Attributes
Status
changed from pending review to published (public).
Published At
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.
Presentation
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Age
changed from 16 to 16 years.
Body
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Fahr syndrome, also called estriatopalidodendate calcinosis, is characterized by extensive calcification of the base ganglia, dentate nucleus, thalamus, and subcortical white matter. It may be primary, familial, or secondary, such as HIV, hypoparathyroidism, hyperparathyroidism, sequelae of chemotherapy or radiotherapy, among others.
-<p>Fahr syndrome, also called estriatopalidodendate calcinosis, is characterized by extensive calcification of the base ganglia, dentate nucleus, thalamus, and subcortical white matter. It may be primary, familial, or secondary, such as HIV, hypoparathyroidism, hyperparathyroidism, sequelae of chemotherapy or radiotherapy, among others.</p>- +<p><a title="Fahr syndrome" href="/articles/fahr-syndrome-1">Fahr syndrome</a>, also called estriatopalidodendate calcinosis, is characterized by extensive calcification of the base ganglia, dentate nucleus, thalamus, and subcortical white matter. It may be primary, familial, or secondary, such as HIV, hypoparathyroidism, hyperparathyroidism, sequelae of chemotherapy or radiotherapy, among others.</p>
Updates to Study Attributes
Caption
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CT Brain
Findings
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Extensive symmetric calcifications involving caudate, lentiform nucleus, thalamus, and subcortical white matter.