Posterior reversible encephalopathy syndrome
Updates to Case Attributes
Cross section imaging findings classical of PRES - with an appropriate associated clinical history.
Posterior reversible encephalopathy syndrome (PRES) (also known as hypertensive encephalopathy) is a neurotoxic state occuringoccurring secondary to the inability of posterior circulation to auto-regulate in response to acute changes in blood pressure. Hyperperfusion with resultant disruption of the blood brain barrier results in vasogenic oedema, most commonly in the parieto-occipital regions.
The blood product sensitive sequences show evidence of hypertensive microangiopathy, indicating an acute on chronic problem with blood pressure.
-<p>Cross section imaging findings classical of <a href="/articles/posterior-reversible-encephalopathy-syndrome-1">PRES</a> - with an appropriate associated clinical history.</p><p>Posterior reversible encephalopathy syndrome (PRES) (also known as hypertensive encephalopathy) is a neurotoxic state occuring secondary to the inability of <a href="/articles/missing">posterior circulation</a> to auto-regulate in response to acute changes in blood pressure. Hyperperfusion with resultant disruption of the blood brain barrier results in vasogenic oedema, most commonly in the parieto-occipital regions.</p><p>The blood product sensitive sequences show evidence of <a title="Hypertensive microangiopathy" href="/articles/hypertensive-microangiopathy">hypertensive microangiopathy</a>, indicating an acute on chronic problem with blood pressure.</p><p> </p>- +<p>Cross section imaging findings classical of <a href="/articles/posterior-reversible-encephalopathy-syndrome-1">PRES</a> - with an appropriate associated clinical history.</p><p>Posterior reversible encephalopathy syndrome (PRES) (also known as hypertensive encephalopathy) is a neurotoxic state occurring secondary to the inability of <a href="/articles/missing">posterior circulation</a> to auto-regulate in response to acute changes in blood pressure. Hyperperfusion with resultant disruption of the blood brain barrier results in vasogenic oedema, most commonly in the parieto-occipital regions.</p><p>The blood product sensitive sequences show evidence of <a href="/articles/hypertensive-microangiopathy">hypertensive microangiopathy</a>, indicating an acute on chronic problem with blood pressure.</p>
Updates to Study Attributes
Periventricular, largely posteriorly located high T2 signal in a symmeterialsymmetrical fashion.
The DWI/ADC maps show increased diffusion.
SWI shows haemosiderin products in keeping with old mircohaemorrhages of hypertension.
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